Figure 4From: A polymorphism in the interleukin-4 receptor affects the ability of interleukin-4 to regulate Th17 cells: a possible immunoregulatory mechanism for genetic control of the severity of rheumatoid arthritisIn vivo interleukin (IL)-17 production in healthy individuals and RA patients. Comparison of control and RA serum IL-17 levels, P = 0.05.Back to article page