Fig. 3From: The impact of dysfunctional variants of ABCG2 on hyperuricemia and gout in pediatric-onset patientsa The influence of p.Q141K: homozygotes develop hyperuricemia very early compared to heterozygotes and wild-type homozygotes. b The existence of familial hyperuricemia/gout shifts the age of onset towards earlier ages in the whole setBack to article page