Fig. 3From: Functional analysis of novel A20 variants in patients with atypical inflammatory diseasesNF-κB reporter gene activity of TNFAIP3 variants using A20-deficient HEK293 cells and the Tet-on system. The expression levels of A20 were controlled by the Tet-on system. The variant E192K could not suppress the increase of activity regardless of the A20 expression level compared with the WTBack to article page