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Fig. 2 | Arthritis Research & Therapy

Fig. 2

From: Elevation of α-1,3 fucosylation promotes the binding ability of TNFR1 to TNF-α and contributes to osteoarthritic cartilage destruction and apoptosis

Fig. 2

The fucosylation level was increased with the development of OA. The OA rabbit model was established to assess the dynamic change in glycopatterns in cartilage during the development of OA. a The X-ray image was used to evaluate the morphology of knee joints from OA models at 0, 4, 8, and 12 weeks after surgery (upper). Cartilage tissue sections were obtained from the right knee joints of OA models and stained with H&E (middle) and safranine-O (lower) for the indicated time periods (original magnification ×100). b The NFIs of 5 lectins were significantly altered in OA models (n=3 per time point) compared with sham operation controls (n=3 per time point) based on fold change and t test. (NS not significantly different, NS no significant difference, *p < 0.05, **p < 0.01, and ***p < 0.001)

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